Sepsis, Neonatal
BASICS
DESCRIPTION
- Clinical syndrome characterized by systemic infection occurring within the first 28 days of life
- Early-onset sepsis (EOS) and late-onset sepsis (LOS) occur within and following the first 72 hours of life, respectively.
- Some consider infection within the first 7 days of life early onset, especially in term infants who are not hospitalized.
EPIDEMIOLOGY
- EOS: 1/1,000 live births in the United States. Incidence is inversely associated with gestational age and birthweight. Highest rates are seen in very low-birth-weight (VLBW) neonates (13/1,000) and neonates born before 28 weeks (18/1,000).
- LOS in the community: 4/1,000 full-term infants presenting to care. Incidence declines with age. Among febrile neonates, approximately 12% are diagnosed with a serious bacterial infection such as bacteremia, meningitis, or, most commonly, urinary tract infection (UTI).
- LOS in hospitalized neonates: Incidence is inversely associated with birth weight and is as high as 20–30% for VLBW neonates.
ETIOLOGY
- EOS:
- Group B Streptococcus (GBS) (~,30%) and E. coli (~30%) are most common, followed by other streptococci (~10%).
- Incidence of GBS disease has decreased since initiation of intrapartum antibiotic prophylaxis (IAP).
- E. coli is the most common pathogen in VLBW infants.
- Overall incidence of Listeria sepsis is low, but it is more common in preterm infants.
- Viral pathogens, including herpes simplex viruses (HSV) and enteroviruses, may present with EOS.
- LOS:
- For hospitalized neonates, the most common pathogens are coagulase negative staphylococci (CoNS) (~50%), E. coli, Staphylococcus aureus (up to 25% methicillin-resistant), and GBS.
- For neonates presenting from the community, the most common pathogens include E. coli (~60%), GBS (~20%), and S. aureus (~5%).
- Pseudomonas aeruginosa carries the highest mortality risk in preterm infants (up to 75%).
- Consider fungal infections; hospitalized preterm infants are at highest risk.
- Viral pathogens, including enteroviruses, may present with LOS; viral meningitis is more common in the late-onset period.
- Perinatally acquired HSV infection should be considered in neonates at any period during the 1st month of age.
RISK FACTORS
- Neonates are at increased risk for infection due to their immature immune system (permitting relative immune tolerance) and their developing, under-keratinized cutaneous barrier.
- Maternal factors:
- Intra-amniotic procedures
- Prolonged (>18 hours) or premature rupture of membranes
- Intra-amniotic infection (IAI) or “intrauterine inflammation, infection, or both” (III, “triple I”); new terms describing the heterogeneous conditions labeled previously as chorioamnionitis
- Presence of cervical cerclage
- Septic/traumatic delivery
- Peripartum infection, including UTIs
- GBS colonization
- Illicit substance use
- Inadequate or no prenatal care
- Poor maternal nutrition
- Ingestion of contaminated foods during pregnancy (Listeria)
- Infant factors:
- Male sex
- Prematurity or low birth weight
- Low APGAR scores
- Congenital anomalies
- Compromised skin integrity
- Galactosemia (Escherichia coli sepsis)
- Invasive procedures
- Presence of central line, indwelling catheters, or endotracheal tube
- Prolonged hospitalization
GENERAL PREVENTION
- Recognize and treat maternal peripartum infections and colonization (i.e., GBS).
- Follow obstetric practices to reduce risk of IAI/III and maintain postnatal clean cord care.
- Maintain effective thermoregulation.
- Encourage early breastfeeding.
- Practice hand hygiene; avoid fomites.
- Minimize unnecessary central line use, and follow evidence-based line insertion and maintenance practices.
PATHOPHYSIOLOGY
- EOS: primarily vertical transmission, with antepartum or intrapartum acquisition of bacteria colonizing the maternal genitourinary tract
- LOS: primarily due to horizontal transmission or nosocomial infection in hospitalized infants
COMMONLY ASSOCIATED CONDITIONS
- Meningitis: Up to a quarter of neonates with bacteremia have meningitis
- Pneumonia
- UTI
- Omphalitis
- Osteomyelitis
- Severe hyperbilirubinemia
- Persistent pulmonary hypertension of the newborn (PPHN)
- Patent ductus arteriosus (PDA)
- Viral infections: The rate of bacteremia in viral-positive neonates is similar to the rate of bacteremia in viral-negative neonates. Similar testing and treatment for neonatal sepsis is warranted for viral-positive neonates, including those with SARS-CoV-2 infection.
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Citation
Cabana, Michael D., editor. "Sepsis, Neonatal." 5-Minute Pediatric Consult, 9th ed., Wolters Kluwer, 2025. Pediatrics Central, peds.unboundmedicine.com/pedscentral/view/5-Minute-Pediatric-Consult/618236/all/Sepsis__Neonatal.
Sepsis, Neonatal. In: Cabana MDM, ed. 5-Minute Pediatric Consult. Wolters Kluwer; 2025. https://peds.unboundmedicine.com/pedscentral/view/5-Minute-Pediatric-Consult/618236/all/Sepsis__Neonatal. Accessed June 2, 2026.
Sepsis, Neonatal. (2025). In Cabana, M. D. (Ed.), 5-Minute Pediatric Consult (9th ed.). Wolters Kluwer. https://peds.unboundmedicine.com/pedscentral/view/5-Minute-Pediatric-Consult/618236/all/Sepsis__Neonatal
Sepsis, Neonatal [Internet]. In: Cabana MDM, editors. 5-Minute Pediatric Consult. Wolters Kluwer; 2025. [cited 2026 June 02]. Available from: https://peds.unboundmedicine.com/pedscentral/view/5-Minute-Pediatric-Consult/618236/all/Sepsis__Neonatal.
* Article titles in AMA citation format should be in sentence-case
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T1 - Sepsis, Neonatal
ID - 618236
ED - Cabana,Michael D,
BT - 5-Minute Pediatric Consult
UR - https://peds.unboundmedicine.com/pedscentral/view/5-Minute-Pediatric-Consult/618236/all/Sepsis__Neonatal
PB - Wolters Kluwer
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DB - Pediatrics Central
DP - Unbound Medicine
ER -

5-Minute Pediatric Consult

